Das verborgene Ärztewissen von 1927 über Impfungen, Krebs, Ernährung, Seele, Zucker, Salz, Kaffee usw.
5 Foods & Spices That Can Prevent And Cure Disease
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Cayenne – stärkt die Abwehrkräfte
Gewicht: 0,05 kg
jetzt mit je 520 mg
Nehmen Sie eine oder mehr Kapseln drei mal täglich mit den Mahlzeiten.
Stabilisiert den Kreislauf und stärkt das Herz
Cayenne wird seit Jahrhunderten als medizinisches und kulinarisches Kraut benutzt. Traditionell wird Cayenne benutzt, um den Kreislauf zu stabilisieren, das Blut zu reinigen und um das Herz zu stärken. Er wirkt sich positiv aus bei Arthritis, Blutungen, Durchblutungsstörungen, Bluthochdruck, Diabetes, Geschwüren, Tumoren, unterstützt die Nieren, die Bauchspeicheldrüse, die Lungen und vieles mehr. Es gibt Kräuterspezialisten, die Cayenne für das nützlichste Kraut im ganzen Pflanzenreich halten.
Die Pflanze wurde von Kolumbus und anderen Seefahrern aus Indoamerika nach Europa gebracht, und später auch aus Indien importiert. Der Hauptgrund jener Reisen war die Suche nach den Gewürzinseln im Indischen Ozean. Man fand aber Amerika und damit viele neue Nahrungsmittel und Gewürze, wie die Kartoffeln, Tomaten, den Mais, Paprika und deren Verwandte und den Cayenne-Pfeffer. Botanisch gesehen hat Cayenne nichts mit Pfeffer zu tun und als Gewürz nur die Schärfe gemeinsam. Bei den Criollos von Bolivien und Peru spielen die brennend-scharfen Capsicum-Arten seit jeher eine wichtige Rolle bei der Speisezubereitung, wie auch bei den Mannbarkeitsriten (erhöht den Blutstrom).
Je schärfer der Cayenne ist, um so wirksamer ist er auch. Oft wird Cayenne mit geringer Schärfe angeboten, weil er nicht so teuer ist. Wir glauben aber, dass wir das wirksamste Produkt anbieten sollten, das verfügbar ist. Deswegen bieten wir Cayenne mit der Schärfe 90,000 HU an. Wegen der natürlichen stimulierenden Wirkung von Cayenne bekommen einige Leute ein harmlose brennende Empfindung einige Minuten nach der Einnahme – einige Apfelstücke bringen das normalerweise in Ordnung.
Bemerkungen zur Anwendung
Traditionelle Indikationen: Asthma, schlechter Atem, Blutungen, Krämpfe, grippale Infekte, Blähungen, Gicht, Herzprobleme, inkorrekter Blutdruck, Magen- und Darmbeschwerden, Verdauungshilfe, Problemen mit der Bauchspeicheldrüse, Rheuma, Krampfadern
Wir kennen Cayenne als scharfes Gewürz. Es fördert die Verdauung, lindert Magen- Darmbeschwerden und kann bei Herzproblemen Linderung verschaffen.
Es werden auch Erfolge bei akuten Herzbeschwerden, ja sogar bei Herzattacken gemeldet. Im akuten Fall vermögen 2-3 Cayenne-Kapseln – oder das Pulver – mit warmem Wasser eingenommen, den Anfall sofort zu lindern.
Regelmäßig angewandt stärkt Cayenne die Abwehrkräfte und lindert Frösteln (ein Kältegefühl am ganzen Körper), da er wärmt und den Blutkreislauf anregt. Cayenne ist reich an Vitamin A, C, PP, B-Komplex, Carotin, Kalzium, Kalium, Phosphor und Eisen. Er gleicht den Blutdruck aus, verbessert die Gerinnungsfähigkeit des Blutes, wirkt antibiotisch und antiviral, fördert den Leberstoffwechsel und den Gallenfluss. Jüngste Untersuchen lassen vermuten, dass Cayenne bei Gürtelrose und Migräne schmerzstillend wirkt. Das Gewürz wird durch das Trocknen und Pulverisieren der Früchte gewonnen. In unseren Breitengraden sollte die Steigerung bis zur Höchstdosis langsam erfolgen und anschließend nur kurzfristig angewendet werden. Beim eventuellen Auftreten einer starken Reaktion bringt ein geraffelter Apfel sofortige Besserung. Schwangere sollten Cayenne nicht in therapeutischen Dosen verwenden. Augenkontakt vermeiden und nicht in offene Wunden streuen.
Cayenne regt den Blutfluss an
Cayenne ist die beste pflanzliche Hilfe für die Blutzirkulation und kann regelmäßig eingenommen werden. Es gibt keine andere Pflanze, die so schnell , stark und vollständig den Blutfluss anregt. Letzten Endes verursachen keine anderen Kräuter ein so rotes Gesicht wie Cayenne – das ist Blut! Es wird für alles genutzt, von Herzattacken, Schlaganfällen, über Ohnmächtigkeit und Schock bis zu inneren und äußeren Blutungen, arthritischen Schmerzen und Entzündungen. Viele Heilkundler schätzen das Kraut sehr und fügen es zu beinahe jeder Formel hinzu.
Eine Krankheit wird immer durch irgendeine Art von Blockade hervorgerufen. Blockaden können im Blut auftreten, in der Lymphe, bei der Sauerstoffzufuhr, bei der Nahrungsaufnahme, bei den Impulsen von Nerven, als auch bei der emotionalen oder spirituellen Energie, welche wir von den Chinesen, Japanern und Indern als Chi, Ki oder Prana kennen. Wenn ein Teil des menschlichen Körpers blockiert ist, stellen sich Probleme ein.
Cayenne ist ohne Zweifel der allerbeste, am meisten zu empfehlende und kraftvollste pflanzliche Entblocker.
We all sometimes wake up in the middle of the night. According to a study, 42% of the people in America are waking up in the middle of the night. Many people want to get rid of this problem.The stress is the real cause for this. A biochemical reaction between the stress hormones occurs when your body rests.
If you have problems with insomnia, the following remedy will help you to solve this problem. By only using salt and sugar you can calm your hormones and fall asleep again.
Many people aren’t aware that these ingredients are crucial for maintaining a healthy metabolism. They regulate the stress hormones and allow your cells to generate energy.Sugar and salt are charging the cells: glucose is a fuel for the mitochondria while a balance of sodium in the intercellular fluids allows proper respiration and energy generation.
Sugar signals the organism to stop with the production of the stress hormones. Salt keeps a homeostatic state in which adrenaline is controlled all the time.
Moreover, the sugar doesn’t cause hyperactivity, as you have already heard.
How to make sleepy dust?
- 1 tablespoon of sucana/rapadura (it is unrefined sugar cane juice) tablespoonHy
- 2 tablespoons of real sea salt
“The salt and sugar mixture is an absolute must for nighttime stress events.For wakeups between 2-4 am, accompanied by a feeling of excess adrenaline circulating through your system (adrenaline peaks at this time), salt and sugar under the tongue is the only way to go. You don’t want to be chewing anything, wandering around the house looking for food, opening the fridge and looking at bright lights when hoping to fall back asleep, and so forth. You want to remain as unstimulated as possible. Keep the sugar/salt mixture by the bedside for easy and thoughtless access until you stop having middle-of-the-nig
How to make sleepy dust?
- 5 tablespoons of organic cane sugar
- 1 tablespoon of sucana/rapadura (it is unrefined sugar cane juice)
- 2 tablespoons of real sea salt
“The salt and sugar mixture is an absolute must for nighttime stress events.For wakeups between 2-4 am, accompanied by a feeling of excess adrenaline circulating through your system (adrenaline peaks at this time), salt and sugar under the tongue is the only way to go. You don’t want to be chewing anything, wandering around the house looking for food, opening the fridge and looking at bright lights when hoping to fall back asleep, and so forth. You want to remain as unstimulated as possible. Keep the sugar/salt mixture by the bedside for easy and thoughtless access until you stop having middle-of-the-night wakeups.”
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Quabain Mother tincture , (Strophanthus gratus) 120ml
Strophantin Mother tincture , Ouabain, Quabain, Strophantus gratus , 120ml
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“All truth passes through three stages. First, it is ridiculed. Second, it is violently opposed.
Third, it is accepted as being self evident”, Arthur Schopenhauer
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strophantin, Strophanthin, Strophantus gratus, strophantus gratus, strodival, kombetin, Ouabain, Acocantherin, Astrobain, G-Strophicor, Gratibain, Gratus, Strophanthin, Kombetin, Purostrophan, Rectobaina, Solufantina, Strodival, Strophalen, Strophoperm, Strophosan, Uabaina, Uabanin, Estrofantina
3 times a day 10 drops on the tongue.
You also can mix it with water.
Let it in the mouth for about 2 to 3 minutes.
Absorption works by mouth mucosis.
Only swallow after this period.
Emergency : 20 to 25 drops
Coronary artery disease is currently the leading cause of death in the United States. Despite the increasing sophistication of surgical techniques, the introduction of new techniques such as balloon angioplasty, and a number of new drugs (e.g. beta blockers, calcium antagonists), it is estimated that over 1 million heart attacks will occur this year, resulting in 500,000 deaths. In short, we do not have an adequate therapeutic solution to the problem of myocardial infarction (heart attack).
The cornerstone of therapy for treatment and prevention of myocardial infarction is to remove blockages in coronary arteries that are thought to be the cause of the infarction. This adheres to the widely accepted coronary artery thrombosis theory of infarction; that is, arteries become clogged with plaque, damaged from such things as smoking or high cholesterol. A clot forms a fissure in the plaque. The clot may shut off the blood flow of the coronary artery, causing a heart attack. It is deceptively simple: The coronary arteries are clogged. No blood can flow, so the muscles of the heart cannot be supported, and heart metabolism stops, leading to death.
In Germany, another theory of myocardial infarction has been proposed by Dr. Berthold Kern (1911-1995). Dr. Kern, while performing autopsies in Germany in the 1930s and 1940s, observed that the findings of these autopsies did not corroborate the coronary obstruction hypothesis. He began researching the literature, looking for clues as to an alternative etiology. What he found was not only a new theory that may provide the missing piece of the coronary obstruction theory, but a therapy now being used by over 5000 physicians in Germany with reportedly remarkable success.
Dr. Kern’s claims, as set forth in his 1971 informational paper, Three Ways to Cardiac Infarction, can be summarized as follows:
1. The coronary obstruction theory cannot adequately explain observed facts.
2. The major etiologic factor underlying myocardial infarction is a primary chemical destructive process, cause by unchecked metabolic acidosis (accumulation of acid) in the left ventricular tissue and substantially unrelated to coronary artery disease.
3. The regular, clinical use of oral g-strophathin (a cardiac glycoside derived from the West African plant strophanthus gratus):
- Prevents lethal myocardial tissue acidosis, and thereby
- Substantially reduces the incidence of myocardial infarction and completely prevents infarction deaths.
Dr. Kern’s observations that most myocardial infarctions occur in patients without significant obstruction of the coronary artery supplying the infracted tissue finds great support in the American peer-reviewed literature. Since 1948, over a dozen reports of post-mortem examination of infracted hearts have consistently failed to corroborate the coronary artery thrombosis theory of myocardial infarction. That is, victims of fatal heart attacks have had no evidence whatsoever of coronary occlusion.
An example of the degree of non-confirmation can be ascertained by the following quote from a 1980 article on Circulation:
“These data support the concept that an occlusive coronary thrombus has no primary role in the pathogenesis of a myocardial infarct.” The reviewer went on to note, “These reports also present clear refutation of the most common explanation used today to dismiss autopsy findings which detect no coronary thrombi, i.e. that thrombi existed at infarction but have since lysed, embolized or washed away.”
There does not appear to be any literature that effectively refutes these autopsy findings.
Another source of inconsistent data are the many reports in the literature of myocardial infarction in patients without coronary artery disease, as deduced by normal coronary angiograms. Other autopsy data has revealed widely scattered areas of necrotic tissue that produces a substantial incongruence between the area of infarction and the arterial supply.
In a 1988 editorial published in the New England Journal of Medicine titled “Twenty years of coronary bypass surgery,” Thomas Killip observed that “Neither the VA [Veterans’ Administration] nor CASS [the National Institute of Health’s Coronary Artery Surgery Study] has detected a significant difference in long-term survival between the two assigned treatment groups [surgical vs. medical] when all patients have been included…”
More recent work with coronary angioplasty and anti-thrombolytic agents has also failed to demonstrate any clear cut improvements in survival.
Dr. Kern went a step further. In his review of the literature, he came across the notion of collaterals (or anastomoses), a finely-meshed network of small blood vessels that act as natural bypass channels in the heart muscle. These collaterals have been made visible by Professor Giorgio Baroldi in studies at the Armed Forces Institute of Pathology.
Baroldi developed a technique for filling the arteries of the heart with artificial blood, a chemical substance that thickens in the blood vessels. When later the tissues were dissolved in acid, the entire structure of blood vessels in the heart was revealed. Kern hypothesized that bypass grafts were created naturally by the body via the collaterals whenever a coronary artery became blocked. Therefore, heart bypass would be redundant to a large degree.
A study by Rentrop et al in the April 1, 1988 issue of The American Journal of Cardiology has produced results completely at odds with the coronary artery blockage theory, and consistent with Kern’s hypothesis. In an accompanying editorial, Dr. Stephen Epstein of the National Heart, Lung and Blood Institute summarizes Rentrop and colleagues’ “extremely important observations.” They found that in an advanced state of the narrowing of the coronary arteries, the supply of blood to the heart muscles is fully assured via collaterals that enlarge naturally in response to the blockage. Interestingly, they observed that the more the coronaries narrow, the less danger there is of heart infarction.
Dr. Kern’s second claim, i.e. his proposed new theory of metabolic acidosis, can be summarized as follows: Metabolic conditions in the most healthy of hearts are, at best, marginal in the constantly beating left ventricle. This is the part of the heart responsible for pumping blood to most of the body, the right ventricle merely supplying the lungs. Oxygen and energy requirements are always perilously close to available supplies, and any of the several stressors may cause an oxygen/energy deficit, with deterioration in oxidative metabolism, and consequent development of acidosis. Lack of oxygen sets off the process of zymosis or fermentation metabolism, an anaerobic process, in order to produce energy in the cells. This, in turn, lowers the pH.
This lowering of the pH sets off a destructive chemical process, literally a suicide reaction of the cell. Lysozymal enzymes are released, causing cell self-digestion. This starts as a single point in the muscle, then many points, which eventually join to form a small area of necrotic tissue. Finally, a critical mass is reached, no bigger than the head of a pin, which triggers larger and larger areas of damaged tissue, resulting in infarction (heart attack).
Ideally then, the remedy to address infarction would be a restoration of pH balance to the heart muscle, thereby preventing tissue damage and fatal infarction. The problem Kern faced was how to accomplish this without causing positive inotropy [increasing the strength of the muscular contraction], i.e. without putting further stress on the contracting heart muscle itself. The cardiac glycosides, including digitalis and the strophanthin byproduct known as ouabain, are known to produce such a deleterious effect, and this is why they are not effective against infarction.
This is where Kern made an important re-discovery. In reviewing the literature, he came across the work of Dr. Edens, who in the 1920s had reported on a qualitatively different effect of strophanthin given intravenously versus orally. Specifically, the positive inotropic effects [that is, increasing contraction] that accompanied intravenous administration were not observed with oral administration.
This important observation has been confirmed in a study by Belz published in theEuropean Journal of Clinical Pharmacology in 1984. Utilizing a randomized, placebo-controlled, double blind methodology, the researchers found that the intravenousouabain (strophanthin) produced the expected increase in cardiac inotropy. However, the investigators stated quite definitely that, “… the single sublingual (oral) dose of ouabain did not exert a positive inotropic effect.”
The postulated mechanism of action, based on animal research done by Adams, Powell and Erdmann, is that there are two receptors in the heart: “High affinity” and “Low affinity.” It is thought that intravenous administration triggers low affinity receptors, and thus positive inotropy. High affinity receptors, on the other hand, react to small concentrations of g-strophanthin via oral administration, thereby avoiding the dangerous effect of positive inotropy.
Dr. Kern reported results of his clinical practice in Stuttgart over the period 1947-1968 involving over 15,000 patients. His patients treated with oral g-strophanthin experienced no fatal infarcts and only 20 non-fatal heart infarcts. These patients included many suffering infarction prior to entering the study. In contrast with these results, government statistics for the same time period would have predicted over 120 fatal heart attacks and over 400 non-fatal infarctions in a group of patients this size.
Currently, there are approximately 5000 M.D.s in Germany using and prescribing oral g-strophanthin. The booklet Eine Dokumentation ambulanz-kardiologischer Therapie Ergebnisse nach Anwendung oralen g-strophanthin represents the results of a survey wherein 3645 medical doctors made statements on use of this remedy in their practices from 1976 to 1983. Of these, 3552 gave exclusively positive testimony with no reservations. No one gave a negative response.
In addition to accumulating clinical experience, a number of studies have demonstrated excellent results with oral g-strophanthin. One fascinating report in a real-life setting took place at a German coal mine. During the period 1972-1974, miners suffered episodes of acute chest pain 229 times. Medical help was a two-hour ride away, and 11 miners died during this period. From 1975-1980, all miners who experienced acute chest pain (280 episodes) were immediately given oral g-strophanthin. During this period, which was twice as long as the comparison period, no miners died after the onset of symptoms. No toxic side effects were observed. Many variables were studied, i.e. age better access to treatment, different working conditions, etc to ensure comparability of observation periods.
A rigorous, double blind, randomized control study of oral g-strophanthin in the treatment of angina showed impressive results at statistically different levels. After fourteen days, 81% of patients in the treated group experienced a reduction in attacks, while in the control group, 72% receiving placebos registered an increase in attacks.
In a study of 150 seriously ill heart patients, who altogether had 254 heart attacks, oral g-strophanthin was successful in 85% of the cases. Dr. Dohrmann, who conducted the study, observed, “A positive result was registered when the severe heart attack abated at least five minutes after the g-strophanthin capsule was bitten through, and after ten minutes at the latest, they disappeared completely.”
A consistent feature of clinical reports using oral g-strophanthin is the absence of side effects. The cost of this remedy, which is currently available to German physicians and their patients, is approximately $30 per month for typical use.
At this point, every indication suggests that oral g-strophanthin may be a significant breakthrough in the treatment and prevention of myocardial infarction. What is needed is a definitive American clinical trial.
At an annual meeting of the American College of Cardiology in New Orleans, it was mentioned that every year one million US citizens suffer a heart attack. Of these, about 60 percent get to the hospital alive. About 16 percent never leave the hospital, and a further 10 percent die within a year. This should be keen motivation for a complete and intensive investigation of the benefits of g-strophanthin.
The prospect of replacing heart bypass surgery with a safer, more effective, and less expensive treatment may be another reason to interest other parties in funding American research on oral g-strophanthin.
STROPHANTUS – QUABAIN
Ouabain – the optimal solution for the problem of myocardial infarction (Extracts from the book “Ouabain – the possible victory over the myocardial infarction” by Rolf-Jürgen Petry*) One of the most necessary things in the contemporary medicine is to call attention to a topic that seems to be unbelievable at first sight: Ouabain (in german: g-Strophanthin), an extraction of an african plant called „strophanthus gratus“, which since 1991 is discovered as an endogenous substance – a new hormone-, prevents angina pectoris and myocardial infarction by nearly 100 percent without side effects. There is an overabundance of studies and documented experiences, so that its effects are quite obvious, even if the great clinical double-blind study is missing. But there is a mighty inscrutable opposition against the therapy with orally administered ouabain. There are two wrong tenets creating an impermeable wall:1) Ouabain is like digitalis classified as a heart glycoside, with the indications „heart insufficiency“ and „arrhythmia“. Because digitalis has negative effects in angina pectoris and myocardial infarction, and „the story goes“ that all glycosides act similarly, the outstanding therapeutical results of ouabain don´t attract any attention at all in the medical establishment.2) In the textbooks is written that ouabain has a very bad oral absorption – but there are over 20 studies which prove the contrary. The worldwide best resultsThe best example for the indeed excellent therapeutic results of oral ouabain in angina pectoris and myocardial infarction is Prof.R.Dohrmann from Berlin (West), who has been the leader of a public hospital, starting 1975 with this therapy. In 1984 Dohrmann & Dohrmann published a study (1) dealing with oral ouabain therapy in unstable angina pectoris. 148 patients with severe stenosis visable in coronary angiography, who received for years all the medicaments modern medicine offers and who are dissatisfied because of continous heart attacks and in part unpleasant side effects, have been switched over (with their agreement after an information discourse) to the therapy with oral ouabain from one day to the other, i.e. the other medicaments including the ß-blockers (!) were discontinued immediately. From these 148 patients 122 were free from angina pectoris after one week, and after two weeks this success could be seen with 146 patients. They were also free from the side effects of the former medication. The other two patients had to stop the therapy because of some irritation of the digestion tract, the only harmless side effect which sometimes occures. (Perhaps a conversion to natural food could help in these cases.) The capsula which is dissolved in the small intestine is sufficient in most cases, and when in spite of this prophylaxis there is a heart attack, the capsula for lingual absorption surely helps in 5-10 minutes in almost every case. With this method every patient can help himself even in the case of acute myoardial infarction, before the ambulance can be present.By the way it is not a necessary condition to discontinue the former medication: there have never been any interactions between oral ouabain and any other medicine, even not with digitalis, on the contrary the experience shows that oral ouabain reduces the side effects of digitalis, when this drug is necessary because of tachycardia.The study of Dohrmann et al. of 1977 (2) deals with sublingually applied ouabain to all patients coming to the hospital with severe heart attacks, acute myocardial infarction (AMI) or suspected AMI. In 170 of 264 cases (= 64 %) the attack was totally stopped within 5-10 minutes, in the rest there has just been an AMI in a relatively late phase in 55 cases, when there is no success to be expected any more, so that only in 15 % of the patients with A.p. (but without AMI) there was no positive result with the first application of ouabain. The above mentioned study (1) shows that the optimal success is reached only within some days.In the therapy of AMI Prof. Dohrmann introduced a new therapy (3) with i.v. cortison to stabilize the lysosomal membranes and i.v. k-strophanthine with great success, although the optimal time for applying ouabain or k-strophanthine in the first minutes of AMI had often just passed by when the patients were reaching the hospital. The quota of nonsurvivors (30 days) after myocardial infarction was previously very high (38 %) because there have been much more elderly people than in (the rest of) Germany. With this therapy he reached the worldwide best rate of survival of that time (in the first year (1977) 17 % nonsurvivors, and 1987, after 10 years, 15 % with experiences with over 1000 patients). A multicenter study of northern Germany reported a quota of 26 % mortality in a comparable period (3 a). Prof. Dohrmann was outnumbered only by Prof.DeMesquita from Sao Paolo (4) who used ouabain i.v. (9%).Another example is a coal mine in Gelsenkirchen/Germany (5) where the average number of workers dying because of AMI in the mine under the surface of the earth was 3 every year; the way to the doctor lasted more than half an hour. After the doctor of the mine in 1974 began with oral ouabain therapy directly in the mine, the mortality concearning AMI was reduced to zero in the following 10 years.In 1984, the small firm “Herbert Pharma” (the former producer of Strodival® – the only available ouabain medicament nowadays – in Wiesbaden / Germany) had made an inquiry to 3650 doctors with respective experiences (6). Ca. 98,5 % answers were very positive, and 1,5 % were positive with some limitation. Reading the released extract, the answeres of 300 physicians, published with full adress, is really convincing, very often they say: “excellently effective”, “no side effects”, “better than the rest”, “I don’t see deadly myocardial infarctions any more” and so on… (There wasn’t enough money to corrupt such a great number of physicians to make such definite statements. The author talked to some of them, so that it wasn’t a faked inventory)The author could motivate some physicians to use ouabain. Their reports of a good therapeutical success are a good current authentication of the findings in the literature. The anthroposophic “Ita Wegman Klinik” in Switzerland are using ouabain since 2002 with the expected success. There are still about 3000 physicians in Germany who are using oral administered ouabain. Ouabain and Digitalis behave oppositely at the cellular levelThe commonly accepted receptor for heart glycosides, i.e. ouabain and digitalis and some other substances, is the sodium pump, which is present in the wall of every cell in a great number and which is pumping Natrium out of the cell and Kalium into it. This is very important for many fundamental functions of the cell.In all textbooks and articles there is written that heart glycosides are inhibitors of the sodium pump. Ouabain is extensively used in many scientific experiments to block the sodium pump. But for all that, this is more false than true: The inhibition of the sodium pump occurs only with high concentrations of ouabain. On the contrary, the low concentrations of ouabain, which are present in the human body after taking the medicine or naturally because of the endogenous nature of ouabain, have the opposite effect. There are over 50 unnoticed and unrefuted studies, that report of the stimulation of the sodium pump by low doses of ouabain. The last one is Gao et al. 2002 from the University of New York (7). The result is a reduction of sodium and because sodium and calcium are associated (sodium-calcium-exchanger), also a reduction of calcium intracellularly. For digitalis the data is poor, but it seems to be that digitalis is not able to stimulate the sodium pump. This is the explanation for the differences between ouabain and digitalis preparations seen in many pharmaco-dynamical studies and especially seen in the therapeutical results.Two study groups have detected an new receptor for heart glycosides in the inside of the cell, at the sarcoplasmic reticulum, the calcium store of the cell (8-9). The effect is a release of calcium. Ouabain acts tenfold weaker than Digoxin at this new intracellular receptor. Furthermore Santana et al. in 1998 (10) could show, that 0,1 nanoMol of digoxin, a concentration below those that are found in the human blood after digoxin medication, have the halfmaximal effect of opening the sodium channels for calcium, letting in 30 percent of the total calcium influx, whereas ouabain needs nearly the hundredfold concentration to achieve this effect. These differences between the actions of ouabain and digoxin are a good additional explanation for the different therapeutical effect of ouabain and digitalis in angina pectoris and heart infarction.The fact that the activity of the sodium pump of heart cells in ischemia is diminished, and all scientists agree that the sodium pump plays the key role in the fate of these cells in this situation, is the background for the undoubtly excellent, unparalleled and solidly documented therapeutical effect of orally administered ouabain in angina pectoris and myocardial infarction. Ouabain affects several componentsBecause the sodium pump is present in every cell, ouabain can influence not only the heart muscle cells. Because the cells exchange a part of their sodium for calcium, ouabain acts on the whole like a calcium antagonist. In addition ouabain combines the qualities of a whole train of medicaments.The nerve cells as well have sodium pumps, they are even the cells with the highest frequency. Sharma et al. in 1980 saw a reduced output of norepinephrine in nerve terminals with low doses of ouabain, no change with intermediate concentrations and the normally published increase with high concentrations of ouabain (11). Gutman and Boonyaviroj 1977 (12) report that only low doses of ouabain are reducing the output of epinephrine and norepinephrine in the adrenal glands. So the medicament ouabain lowers the level of stress-hormones in the whole body and especially in the myocardium and acts also in the field of a beta-blocker.Ouabain is enhancing the blood flow in the myocardium by action on the blood vessels and on the red blood cells. Manfred von Ardenne, the great scientist of the German Democratic Republic, made many investigations dealing with ouabain, for example in 1991 a scintigraphy with 99technetium showing a marked increase in myocardial blood flow by orally administered ouabain in patients with angina pectoris (13). This was confirmed by the findings of Vatner & Baig 1978 (Harvard) with ouabain i.v. in an animal experiment with dogs (14). Von Ardenne wrote one of the very few articles in English language about the therapy with orally administered ouabain (15).Ouabain is also lowering the blood pressure – but only in the case of hypertension. A good example for the effect of low doses of ouabain on vascular smooth muscle is a study of Branco and Osswald from 1986 (16), where the authors report of the different actions of three different concentrations of ouabain on dog blood vessels. The two high concentrations caused a constriction (release of norepinephrine, a stress-hormone), but the low concentration had the contrary effect, as it would be expected from the studies of DeMots et al.1978 (17) and Marjorie et al. 1997 (18). DeMots et al. 1978 (17) gave the same doses of ouabain i.v. to patients with different velocities. As expected, only the slow injection of ouabain caused a reduction of blood pressure and lactate levels in the heart.Particularly important is the effect of ouabain on the red blood cells. Their diameter (8 micrometers) is bigger than the diameter of the capillaries, which they must pass (3 micrometers). To manage this, the erythrocytes have to make themselves very thin and long, really like a submarine. But because the acid and the free radicals generated in ischemia or even by an overactivity of the sympathetic nervous system (stress hormones) reduce the activity of the sodium pump of the erythrocytes, too, they become full with sodium and therefore full with water and tight and unflexible. Low concentrations of ouabain are able to stimulate the sodium pump of the erythrocytes. Prof.Lohmann (Giessen/Germany) in 1986 found a marked improved flexibility of erythrocytes even at low pH (19). So they get slim enough to pass through the capillaries again. Here Ouabain acts principally like an AAS preparation, like Aspirin®. Prof.E.Ernst and Dr.T.Saradeth 1991 (20) from Vienna had made a randomized, double-blind and placebo-controlled crossover-study with oral ouabain and found significant beneficial effects regarding the erythrocytes and hemodynamic parameters, for example a reduced rise of diastolic blood pressure in exercise.Otherwise the situation could get very harmful. Because of the reduced flexibility of the red blood cells, the bloodflow is reduced and the acid accumulates. That will make the erythrocytes even more immobile and so on – a real vicious circle, that can produce the death of single heart muscle cells or even of bigger regions. This is a widely unnoticed mechanism of how a myocardial infarction could be generated. The reduction of the discussion on the formation of a coronary thrombus is not useful, because there are many studies that report of a low frequency of coronary thrombosis and in addition the thrombosis is time-dependent. The longer the period between myocardial infarction and death of the patient, the bigger is the frequency of coronary thrombosis. The well-known Swedish cardiologist Erhardt (et al. 1973 and 1976) made the final prove: He injected a portion of radiactive labeled fibrinogen into patients after a myocardial infarction. Fibrinogen builds up the thrombus and after this formation there is no exchange of fibrinogen any more. When a patient had died and a thrombus was found, in every case (!) the radioactivity was present in the whole thrombus, even in the middle section (21-22). This implies that the thrombus was not the cause, but the consequence of the myocardial infarction. The fact, that there is a tailback of blood in the coronary system and that in the situation of a myocardial infarction the heart cannot build up the normal pressure from outside into the coronary system, facilitates the formation of a coronary thrombosis at a point of a coronary artery which is narrowed formerly. So the action of ouabain on the red blood cells is very important.In the last years the theory of the vulnerable coronary plaques has become more and more important. In the study of Matsumori et al 1997 (23) ouabain caused the quadruplication of survival in rats with sepsis because of a suppression of cytokine production ( IL-6 and TNF-alpha). Perhaps ouabain could show this effect also in the heart and coronary system and reduce the inflammatory process in the instable coronary plaques – this perhaps is another explanation for the beneficial therapeutical effect of low doses of ouabain. Generally speaking, ouabain acts like a nitro-preparation:There is a randomized, placebo-controlled double-blind study – Salz & Schneider of 1985 (24) in seven doctor´s surgeries which shows an excellent and highly significant result. The classical double-blind experiment of Kubicek and Reisner of 1973 (25) with angina pectoris-patients under hypoxia showed in 19 of 22 patients a marked improvement of the electrocardiogram (S-T-alterations) – in 7 cases a total normalization – after oral ouabain application in comparison to a control group, and the result of subjective state of health is as follows: control: 18 patients with pain or giddiness and only 4 without trouble. After oral ouabain: Only 4 patients with pain or giddiness and 18 patients are without trouble (!). A placebo showed no effect. Digitalis (several drugs in differentiated doses) had a negative effect, so that some experiments had to stop before the regular end. (Reference unfortunately only in German) Also Sharma et al. 1972 (26) had similar good results with i.v. ouabain. This is the corroboration of the therapeutical results reported by Prof. Dohrmann and others, see above.Prof. Belz et al 1984 (27) made a placebo-controlled double-blind crossover-study, which shows that orally administered ouabain has a constant and significant (in part highly significant) effect on the heart contractility of healthy volunteers that is different from the effect of ouabain i.v. and similar to that of nitroglycerine. This finding was confirmed by a study of Prof. Dohrmann & Schlief-Pflug in 1986 (28) who investigated patients with severe CHD and instable angina pectoris.As we have seen, ouabain combines the actions of several medicaments without their side effects and is the perfect pharmacological solution in the case of angina pectoris and myocardial infarction.What is the effect of low concentrations of ouabain in ischemia/reperfusion injury? I never have seen a study in this direction at all. I think such an investigation could be a breakthrough ! Gousious et al. in 1967 made a study (29) which investigates the influence of ouabain in the isolated heart without ischemia: there was an increase in fatty acid oxidation in absence of increased uptake. The study of Horackova and Mullen in 1988 shows a reduction of the Ca++ content of isolated cardiomyocytes with low doses of ouabain, unchanged results with intermediate and the normally published rise with high concentrations of ouabain (30). Ouabain as a possible universal remedyThere are 30 diseases other than angina pectoris, myocardial infarction and heart failure, for example diabetes, cancer, multiple sclerosis, Morbus Parkinson and Alzheimer, seizure disorder, schizophrenia, obesity, inflammable intestinal diseases, arthritis, allergies, toxications,…, in which the activity of the sodium pump of the regarding tissues or of the red or white blood cells is lowered. In this cases non-parenteral ouabain hypothetically could have a positive influence. There are no studies regarding the effect of ouabain in this diseases up to now. The diseases in which a therapeutic effect of ouabain is documented are cerebral ischemia, asthma bronchiale and endogenous depression (31-33). The false dogma of the bad absorption of oral ouabainThe opinion that ouabain is very poorly absorbed when enteral administered, is not valid. Marzo et al. in 1974 (34) could demonstrate, that radioactively labeled ouabain (3H-ouabain) was absorbed at a rate of 36 % within 5 hours in guinea-pigs. Greenberger et al. in 1969 (35) found in the intestine of guinea-pigs for 3H-ouabain a mucosal tranfer of 19 % after only 30 minutes, more than 3H-digoxin (15 %). Forth et al. (36) reported of an enteral absorption of 3H-ouabain after only 1 hour at a rate of 24 % and 48 % in rats and guinea-pigs, respectively. There are some other examples (37-43): Kitano et al. in 1998 (44) refer of a high absorption of orally administered ouabain to rats, comparable with that of digoxin.The last study to mention is Leuschner & Winkler in 2001, who found 50 % of the orally administered 3H-ouabain in the organs of guinea pigs (45).Lauterbach suggested that there is an active transport process for polar cardiac glycosides like ouabain through the intestinal cells (46-47). As a parallel there is an uptake process per endocytosis into the intracellular compartments of the myocardium (48).There are high and non fluctuating blood levels of ouabain in humans in the studies of Erdle et al. in 1979 (49) and Marchetti et al. in 1972 (50) using orally administered 3H-ouabain, which astonishingly are not mentioned in the “Handbook of Experimental Pharmacology” (51). Here only the third respective study of Lahrtz et al. in 1968 (52) is mentioned, in which 1) too little of ouabain was given (0,04 mg; a normal therapeutic dose is 3-12 mg) and 2) indeed there was given too little of radioactivity, even below the detection limit, so that a positive result was impossible even if there had been an i.v. application. However, also the effects of orally administered ouabain on human haemodynamics support the finding of a high and linear, not uncertain absorption (Piscitello &b Maggi in 1973) (53). Interesting are also the studies of Belz et al. in 1984 and Dohrmann at al. in 1986 (27, 28).Investigations with the RIA-method show much smaller amounts of ouabain-immunoreactivity in human blood after oral application, but also here is visible that there is no uncertain absorption, because the fluctuation of perlingual ouabain blood levels is less than the fluctuation after i.v.application (54-55), see below.The fact that in the case of heart infarction there are no high levels of ouabin necessary like in the case of heart insufficiency, is unknown. Prof.Bereiter-Hahn (Frankfurt) in 1991 could show that even a concentration of 10(-13) M (= 60 quadrillionth gram in one millilitre) of ouabain has a reproducible effect on cardiomyocytes regarding the oxygen metabolism, in some cases even a concentration of 10(-15) did so (56). Another study (57) of the same group shows that 10(-10) M ouabain increases markedly the fatty acid oxydation of cardiomyocytes (but not the oxydation of other substrates), which is markedly diminished in ischemia. Only a hair raising inconsistancy or a prearranged deception ?The often published statement of the uncertain enteral absorption of ouabain has its only root in a single examination of another ouabain preparation called Purostrophan ®, which was available in the 1970ies and showed indeed some fluctuation of the results concerning the blood and urine levels of ouabain. Beside the fact, that this fluctuation wasn´t bigger than that of the commonly used Digitalis medicaments, I discovered, that the real examination of oral Purostrophan wasn’t made in the oftenly cited study (54) of Prof. Greeff et al. in 1974, the accepted authority at that time, but one year before in a dissertation of his institute (58). The peculiarity of this dissertation is, that the examination of Purostrophan® has been made with two different groups of patients: one group took ouabain before and the other group took ouabain after the breakfast. In the text of the dissertation the expected result is written: The group who took the medicine before the breakfast had absorbed more ouabain than the other group. Already in the dissertation the different results were summarized to one diagramm and then were repeated by Prof. Greeff in his study (54) and other articles without citation, so that the origin remained hidden. This is the origin of the intensively published statement of an uncertain absorption of ouabain. This nonscientific results are published in the same study that reports of the lowly enteral absorption of ouabain and influenced the majority of the physicians not to prescribe ouabain to their patients. The detection of ouabain as a new hormone and the birth of a new false dogmaSince 1991 ouabain is identified as an endogenous compound, produced by the adrenal glands and / or by the brain (hypothalamus). By the way, the possibility that ouabain is taken up with the food cannot be excluded – this would be another proof for the good absorption of ouabain. Unfortunately here you can observe the birth of a new false tenet: the role of endogenous ouabain as the cause of hypertension, which is based on contradictory studies with rats. The studies with other animals and one double blind clinical study are reporting of an unchanged or even lowered blood pressure, as do other studies and reports. For example, Tamura et al. in 2000 (59) report that a synthetic diet completely without heart glycosides causes hypertension in rats, which is prevented by orally administered ouabain in low dosage.One of the studies, which deals with ouabain as the cause of essential hypertension, contains a surprising hint for the beneficial therapeutic effects of ouabain: Yuan et al. 1993 (60).. The rats with ouabain showed no cardiac hypertrophy, but the rats in the control group did so. Yuan et al. p.186: “Ouabain actually may be cardioprotective.” The most important sentence of the whole study ! There are older studies who also report of the finding of the prevention of the hypertrophy of the heart and adrenals (61-62). The solution of the problem is not wantedUnfortunately there has been a systematic opposition against the excellent therapy with oral ouabain in angina pectoris and myocardial infarction, a German “speciality”, so that the very many published convincing proves were not proclaimed adequately. This exasperated controversity reaches back to ca. 1950, when some of the German universities supported this therapy with which you see results which are unparalleled by any other, even modern medication. From 1905 until the 1950ies ouabain i.v. was the official therapy in heart insuffiency and also partially in angina pectoris and myocardial infarction. Especially in the 19seventies and 19eighties an International Society for Infarction Control (= “Internationale Gesellschaft für Infarktbekämpfung”) with many supporting professors (Schaefer/Heidelberg, Baroldi/Milano…) strived for acknowledgement of the respective facts, but the “suppression fraction” of the medical establishment was too strong – in that time Prof. Gotthard Schettler was the main opposer. Prof. Erdmannn, director of the university hospital of Cologne / Germany and scientist with the main focus heart glycosides, was / is(?) the one who fighted against both the ouabain therapy and the stimulation of the sodium pump by low doses of ouabain. The attacks against ouabain were always unsubstancial and truthless, in part they were contradictory to the own statements made elsewhere.The scepticism of many doctors in testing a substance which is classified as a cardiac glycoside, in angina pectoris, especially when in all textbooks is written that there is 0 % or 1-2 % absorption, is huge. There is a doctor, who has seen the excellent success in every of his 120 Angina pectoris-patients, but his collegues are laughing and even don’t want to hear his story to the end. (I hope you are still reading…) There are doctors who have Strodival® in their drawer, and in case of emergency (severe angina attack, suspected AMI) they quickly applicate the capsula, but they don’t inform the hospital, what they have done, because they fear to make themselves ridiculous. A real tragical story…It is an urgent challenge to save the real possibility to solve one of the biggest medical problems (really: Oubain could be in Coronary Heart Disease something like insulin is in Diabetes.) and to preserve a blessed medicine that helps so many patients. But there is an acute danger that oral ouabain could die, especially after an imminent change in the respective law, so that the sick-funds don’t pay any more for it. Even more grave: it is expected, that there is no prolongation of the licence for orally administered ouabain, because the big clinical double-blind study is missing, as it would cost about 10-15 millions of Euro, much more than the producer company could pay. Even a big multinational company would not pay this sum for a substance without a protection by patent. The orally ouabain therapy may die in 2 years. A real tragedy…A special characteristic is the fact that in 1996 the small producer “Herbert Pharma” in Wiesbaden/ Germany was adopted by “BRAHMS Arzneimittel GmbH” in Wiesbaden (the new name was “Herbert Arzneimittel GmbH”). The director of the mother company – “BRAHMS AG” in Henningsdorf (Berlin) is the President of the German Federal Association of Pharmaceutical Industry, Dr. Bernd Wegener. The new producer is noticeable inactive … . In 2003 “Herbert” was selled by “BRAHMS” to “MEDA”, a Swedish combine.Without doubt ouabain (g-strophanthin) could be the solution of the problem cardiac infarction (“Killer No. 1”) to a large extent and especially the therapy with orally administered ouabain and the knowledge about this drug has to be brought forward because of medical, ethical and economic aspects. * About the authorAfter Rolf-Jürgen Petry had finished his schooling as an alternative practitioner in 1997, he became a permanent visitor of the medical libraries, where he had copied about 20.000 pages in the last years about ouabain and myocardial infarction. He has read the original studies in a very accurate form and has found that the orthodox positions, with which ouabain is rejected, exhibit serious faults. It’s really a severe miscarriage of medical justice, as you could see with the proofs above.He has written the first extensive and detailed book on this theme with 1380 references and the preface of Prof. Hans Schaefer from Heidelberg, who was world-famous for some decades. (Rolf-Jürgen Petry: Strophanthin – der mögliche Sieg über den Herzinfarkt. (Ouabain – the possible victory over the myocardial infarction), 286 pages, 21,90 € (Verlag Florilegium 2003, ISBN 3-00-010149-7), Pf 1305, D 27442 Gnarrenburg, Germany, Tel. 0049 – 4763 – 1092 , Fax 0049 – 1212 – 55 14 09 321, e-mail: firstname.lastname@example.orgThe book is on a high scientific level, but at the same time the author had taken great care that it remains understandable even for a person without previous medical knowledge. This book is available only in German language until now.References:1) in german: R.E.Dohrmann & M.Dohrmann: Neuere Therapie der instabilen Angina pectoris bei koronarer Herzerkrankung, Erfahrungsheilkunde – acta medica empirica 33: 183-190, 19842) in german: R.E.Dohrmann; H.D.Janisch & M.Kessel: Klinisch-poliklinische Studie über die Wirksamkeit von g-Strophanthin bei Angina pectoris und Myokardinfarkt, Cardiol Bull (Cardiologisches Bulletin) 14/15: 183-187, 19773) summary in english: R.E.Dohrmann & R.F.Heller: Therapeutische Ergebnisse bei akutem Myokardinfarkt unter Anwendung hoch-dosierter Steroidgaben und fluiditätsbeeinflussender Pharmaka – Ergebnisse einer 10-Jahres-Studie, Cardiol Bull 24: 17, 19873 a) H.J.Avenarius, H.Poliwoda & B.Schneider: Unersuchungen zum klinischen Verlauf des akuten Myokardinfarkts. Med Klin 72: 459-464, 19774) Prof. Quantiliano de Mesquita, Professor Honorario de Faculdade de Medicina de Universidade Federal de Paraiba. 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Br J Pharmacol 108: 1043-1050, 19939) Masako Fujino & Sumiko Fujino: An immunohistochemical study of the significance of a new 31,5 kD ouabainreceptor protein isolated from cat cardiac muscle. Jpn J Pharmacol 67: 125-135, 199510) L.F.Santana, A.M.Gomez & W.J.Lederer Ca++ flux through promiscuos cardiac Na+ channels: slip-mode conductance. Science 279: 1027-33, 199811) Virendra K.Sharma, Lorelle A.Pottick & Shailesh Banerjee (New York): Ouabain stimulation of noradrenaline transport in guinea pig heart. Nature 286: 817-819, 198012) Yehuda Gutman & Punya Boonyaviroj: Mechanism of inhibition of catecholamine release from adrenal medulla by diphenylhydantoin and by low concentration of ouabain (10 (-10) M). Naunyn-Schmiedebergs Arch Pharmacol 296: 293-296, 197713) M.v.Ardenne, W.-K. Mayer, J.Schmidt, G.Rostock & W.Mohnike (Dresden): Klinische Prüfung des perlingual applizierten g-Strophanthin-Präparats Strodival (R) spezial mit Hilfe der 99 mTc-Myospect-Herztomographie. Z Klin Med 46: 667-669, 199114) S.F.Vatner & H.Baig (Harvard): Comparison of the effects of ouabain and isoproterenol on ischemic myocardium of conscious dogs, Circulation, 58: 654, 197815) M.von Ardenne: Research on the mechanism of myocardial infarctions and on counteracting measures, a new galenic form of the fast acting g-strophanthin, Agressologie 19: 13-22, 197816) D.Branco & W.Osswald: Ouabain-induced efflux of catecholaminess and metabolites from blood vessels of normotensive and hypertensive dogs, in E.Erdmann, K.Greeff, J.C.Skou: Cardiac Glycosides 1785-1985, Steinkopff Verlag, Darmstadt, 198617) Henry DeMots, Shabudin H.Rahimtoola, John H.McAnulty, George A.Porter (Portland / USA): Effects of ouabain on coronary and systemic vascular resistance and myocardial oxygen consumption in patients without heart failure, Am J Cardiol 41: 88-93, 197818) H.J.Marjorie G.Nelissen-Vrancken, Ju-Feng Wang, Harry A.J.Struijker Boudier, Regien G.Schoemaker, Jos S.F.Smits: Ouabain improves cardiac functionin vivo in rats with heart failure after chronic but not acute treatment. 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Arzneimittel-Forsch 18: 1597, 196844) Shoichi Kitano, Shigeto Morimoto, Akira Nishibe, Keisuke Fukuo, Atushi Hirotani, Takeshi Nakahashi, Osamu Yasuda & Toshio Ogihara: Exogenous Ouabain is accumulated in the adrenals and mimics the kinetics of endogenous digitalis-like-factor in rats. Hypertens Res 21: 47-56, 199845) J.Leuschner & A.Winkler (Hamburg): Toxicological studies with ouabain. Naunyn-Schmiedeberg´s Arch Pharmacol 363 (4) Suppl.: 139, abstract 544, 200146) in german, abstract in english: Fritz Lauterbach, Günther Vogel & Ingeborg Baumann: Die Abhängigkeit der enteralen Wirkungsquote kardiotoner Steroide von der angebotenen Dosis, Naunyn-Schmiedebergs Arch Pharmak exp Pathol 259: 248-259, 196847) Fritz Lauterbach in Kurt Greeff (ed.): “Handbook of Experimental Pharmacology”, Vol. 56/II, 198148) Harol Nunez-Duran, Laura Riboni, Ernestina Ubaldo, Emilio Kabela, & Laura Barcenas-Ruiz: Ouabain uptake by endocytosis in isolated guinea pig atria. J Am Physiol 255: C479-C485, 198849) in german: H.P.Erdle, K.D.Schultz, E.Wetzel & F.Gross: Resorption und Ausscheidung von g-Strophanthin nach intravenöser und perlingualer Gabe, Dtsch Med Wschr. (Deutsche Medizinische Wochenschrift) 104: 976-979, 197950) G.V.Marchetti, A.Marzo, C.de Ponti, A.Scvalvini, L.Merlo & V.Noseda: Blood levels and tissue distribution of 3H-Ouabain administered per os, Arzneim Forsch (Drug Res) 21: 1399-1403, 197251) K.Greeff & K.E.Wirth: Pharmacokinetics of strophantus glykosides, in Kurt Greeff: Handbook of Experimental Pharmacology, Band 56 II : Cardiac Glykosides, S.56-85, Springer B-Hdlbg-N.Y., 198152) summary in english: H.Lahrtz, R.W.Sattler & P.A.van Zwieten: Über den Blutspiegel und die Ausscheidung radioaktiv markierter Herzglykoside nach deren intraduodenaler Applikation bei der Katze, Z ges exp Med (Zeitschrift für die gesamte experimentelle Medizin) 148: 210-222, 196853) F.Piscitello & G.C.Maggi: Effectiveness of orally administered g-Strophanthin on haemodynamics in cardiac patients, Arzneim.-Forsch (Drug Res) 23: 1546-1547, 197354) in german: K.Greeff, E.Köhler, H.Strobach, E.Verspohl: Zur Pharmakokinetik des g-Strophanthins, Verh Dtsch Ges Kreislaufforschg (Verhandlungen der Deutschen Gesellschaft für Kreislaufforschung) 40: 301-305, 197455) H.Strobach, K.E.Wirth & K.Rojsathaporn: absorption, metabolism and elimination of strophanthus glycosides in man, Naunyn-Schmiedebergs Arch Pharmacol 334: 496-500, 198656) M.Riehle, J.Bereiter-Hahn & B.Boller: Effects of ouabain and digitoxin on the respiration of chick embryo cardiomyocytes in culture, Arzneim-Forsch / Drug Res 41: 378-384, 199157) M.Riehle and J.Bereiter-Hahn: Ouabain and Digitoxin as modulators of chick embryo cardiomyocyte energy metabolism, Arzneim-Forsch 44: 943-947, 199458) in german: E.Verspohl: Entwicklung radioimmunologischer Methoden zur Bestimmung von Herzglykosiden des Digitoxigenins, g-Strophanthins und k-Strophantidins mit Untersuchungen zur Pharmakokinetik des Digitoxins und g-Strophanthins. Inaugural-Dissertation, Düsseldorf 197359) M.Tamura, H.Utsunomiya, M.Nakamura & E.J.Landon (Nashville / USA): Effect of dietary glycosides on blood pressure regulation in rats: Can J Physiol Pharmacol 78(7): 548-56, 200060) Christina M.Yuan, Paolo Manunta, John M.Hamlyn, Shanwan Chen, Erin Bohen, Jane Yeun, Francis J.Haddy & Motilal B.Pamnani (Bethesda, Washington, Baltimore / USA): Long-term ouabain administration produces hypertension in rats. Hypertension 22: 178-187, 199361) in german: E.Moskopf & H.Dietz: Experimentelle u. klinische Untersuchungen über eine zuverlässige orale Strophanthintherapie. Die Medizinische Welt 1955, p. 1375-7762) in german: G.Kuschinsky: Die Verhütung von Erschöpfungszuständen des Herzens. Klin Wschr 24/25 (1947) 502-503
WE SHIP WORLDWIDE !!!
New Divine Healing Codes!
49 22 249 to officially mark you putting your worst nightmare behind you (this might take some action on your part; work with Spirit to help you resolve what is left of the issue, and be sure to take the steps needed to get it complete!)
13 58 372 for spiritual knowledge to be made freely available for the highest good. (this applies both to the individual and to collectives, however one wishes to apply this Divine Healing Code)
Here is a list to the links to the FAQ’s and full and complete, always up to date list of the codes: http://reikidoc.blogspot.com/p/the-archangel-healing-codes.…Doctors With Reiki
Shortlink : http://wp.me/p2wHrN-6hw
Das Misstrauen gegenüber so manchen Hausmittelchen ist bei einigen groß. Kursieren doch inzwischen viel zu viele von diesen vermeintlichen DIY-Helfern im World Wide Web. Doch einige dieser Tipps, Tricks und Alltagskniffe sind tatsächlich viel mehr von Bedeutung, als wir annehmen wollen! So verhält es sich zum Beispiel auch mit der Küchenzutat „Weinstein“ (auch „Weinsäure“ genannt), die von vielen unterschätzt wird!
Normalerweise dient Weinstein zur Stabilisierung von Eiweiß oder Schlagsahne. Doch was das Pulver noch alles bewirken kann, das erfahrt Ihr nun:
#1 Es befreit Dich von Giftstoffen in der Lunge
Flickr / Artizone Du möchtest das Rauchen aufhören, was ja bekanntlich schon schwer genug ist. Und Weinstein kann Dich dabei unterstützen durchzuhalten! Ein Teelöffel, vermengt mit einem Glas Orangensaft jeden Morgen, löst sämtliche Giftstoffe, die sich zuvor in deiner Lunge abgesetzt haben. So wird das Nikotin aus deinem Blutsystem gespült und die Sucht nach dem Glimmstengel hat schneller ein Ende!
#2 Es senkt deinen Blutdruck
Pixabay Die Ursache für hohen Blutdruck kann oft Kaliummangel sein. Mit Hilfe von Weinstein füllst Du deinen Kaliumhaushalt jedoch ganz fix wieder auf! Mische einen Teelöffel davon in ein Glas Wasser. Jeden Abend ein Glas und im Nu’ stabilisiert sich dein Blutdruck wieder auf normale Werte.
#3 Es verbessert deine Hautstruktur
Flickr / DresdenPlaid Vor allem Menschen, die unter Akne leiden, können sich jetzt freuen. Die sauren Eigenschaften von Weinstein können helfen, Akne verursachende Bakterien zu bekämpfen. Trinke dazu täglich ein Glas Wasser oder Orangenschaft, vermischt mit einem Esslöffel Weinstein. Schon bald wirst Du ein positives Ergebnis feststellen können!
#4 Es verhindert Harnwegsinfektionen
Flickr / Dagus2010 Für Menschen, die häufig von Harnwegsinfektionen betroffen sind, kann Weinstein wahre Wunder bewirken. Denn es lindert nicht nur die Symptome, sondern verhindert auch, dass sie erneut auftreten. Denn es stabilisiert die PH-Werte des Urins. Um einen größtmöglichen Effekt zu erzielen, mische eineinhalb Esslöffel Weinstein mit Wasser und einem Schuss Zitronensaft. Jeden Tag ein Glas und Du wirst schon bald eine Veränderung feststellen!
#5 Es lindert die Schmerzen von Arthritis
Flickr / aptmetaphor Weinstein enthält eine Menge Magnesium und hilft deshalb, Schwellungen und Schmerzen zu lindern. Um beim ersten Auftreten von Arthritis Beschwerden schnellstmöglich entgegenzuwirken, schütte etwas Weinstein und Epsom-Salz in ein heißes Bad. Weiche deine Gelenke anschließend 30-40 Minuten im Wasser ein. Schon bald wird es Dir besser gehen!
Mehr über die Zutat Weinstein und was sie alles bewirken kann, erfahrt Ihr im Video:
Wir sind beeindruckt. Nur wenige kennen diese Zutat, doch von nun an, wird man sie wohl ihn mehreren Küchen vorfinden…
Hanfpapier – lässt die Bäume stehen und ist eine umweltfreundliche und nachhaltige Alternative
Hanf oder Holz zur Papierherstellung, das ist keine Frage. Hanf ist eine Wunderpflanze, die die Sonnenenergie am effizientesten nutzt. Hanf braucht…
Hanfpapier – lässt die Bäume stehen und ist eine umweltfreundliche und nachhaltige Alternative
Hanf oder Holz zur Papierherstellung, das ist keine Frage. Hanf ist eine Wunderpflanze, die die Sonnenenergie am effizientesten nutzt. Hanf braucht…
Hanfpapier – lässt die Bäume stehen und ist eine umweltfreundliche und nachhaltige Alternative
Cannabinol (CBN): The Cannabinoid That Helps You SleepCannabinol is known to serve multiple purposes such as aiding in sleep, acting as an anti-bacterial and providing an indication of cannabis freshness.
Many people practice yoga without knowing about Mudras. It’s time for you to start using these hand gestures that stimulate different areas of your brain and promote health! Share these 8 poses with your fellow yoga lovers today.
Cilantro .. Koriander …. Coriandre ….
Hair loss can be treated with a common backyard herb! Use this plant as an easy remedy for thinning hair or balding- I’ve seen this growing in so many wooded areas around my house.
Gesund und lecker salzen, würzen und verfeinern
Über 40 Prozent rein pflanzliches EiweißBio Süßlupinenmehl
Mit Stevia nehmen Sie gesund abSüß aber gesund
Gesundes Süßen von Speisen und GetränkenXucker Premium
Vom Rande des HimalayagebietesGeschmackvoll salzen
Natürlich süßer Nektar der KokosblüteKokosblütenzucker
Ein uraltes Geheimnis wiederentdecktBambussalz
Die verfeinerte Rezeptur aus der NaturWurzelkraft – Bio
Aus kontrolliert biologischem AnbauBio Moringa Pulver
See more at KOPP-VERLAG.DE
In 2008, on a dig in the First Nation’s Menominee Reservation in Wisconsin, archaeologists made a small but stunning discovery: a tiny clay pot.
Though it might not have seemed very impressive at first glimpse, this little piece of pottery was determined to be about 800 years old.
And inside that pot? Something that changes how we’re looking at extinction, preservation, and food storage, as well as how humans have influenced the planet in their time on it.
It’s amazing to think that a little clay pot buried in the ground 800 years ago would still be relevant today, but it’s true! It’s actually brought an extinct species of squash that was presumed to be lost forever. Thank our Indigenous Ancestors! Even they knew what preservation meant. They knew the importance of the future, Is it not amazing that they are affecting our walks of life even to this day?
Here it is! The pot was unearthed on the Menominee Reservation in Wisconsin, where it had laid buried for the past 800 years.
Inside, archaeologists found a stash of seeds. The seeds were probably buried in the pot as a method of storing food supplies. They were determined to be an old, now-extinct species of squash.
Now, seven years after making this stunning discovery, students in Winnipeg decided to plant the 800-year-old seeds… To everyone’s amazement, something grew!
The squash was named Gete-okosomin. It means “Cool Old Squash” in the Menominee language. (Respect to the Science people for respecting the Indigenous people who’s land this was found on, We See Your Good Nature!)
Now, they’re working to cultivate the squash so that it doesn’t go extinct… again.
It may be just a humble squash, but it’s also a symbol of First Nations’ community and history, as well as a fascinating look into how amazing plants can be.
It just goes to show you that plants can be pretty incredible… and that sometimes, history has a funny way of coming back around.
The Wheel of Life really stands out in this instance of history. Our Indigenous roots are strong and very much tied to the land. I was taught once that the people of Turtle Island were keepers of the land, not owners. I feel like this Squash is proof of that teaching.
Originally taken from cfweradio.ca
I made this DIY rub for my neighbour who suffers from arthritis in her knees. It contains the perfect combination of anti-inflammatories and pain-numbing agents. Try a batch of this easy recipe today for natural relief of joint or muscle pain.
Ready to make a soothing muscle rub for back pain that is natural and free from chemicals?
Well, great. Today I am going to share with you a homemade soothing muscle rub that is easy and fairly quick to make at home. Many of these things you may already have in your kitchen. And the few you don’t, you can easily find at a natural food store or coop.
Now you may be wondering why cayenne would be the main ingredient for a muscle and joint rub. Great question!
Why Cayenne Works for Muscle Pain
Capsaicin, one of cayenne’s active constituents stimulates the release of something known as “substance P” in the body. Substance P is released from nerve cells and the inflammatory cells of the immune system. Substance P is most noted for its role in helping the body perceive pain and in stimulating the release of various cytokines to induce a healthy inflammatory response.
Cayenne also dilates blood vessels, which allows for more anti-inflammatory cells to make their way to the affected area. When substance P is released and subsequently depleted, as is the case when you repeatedly apply cayenne, the threshold of pain in that area increases. This is also known as pain desensitization. 1,2
What happens is the area actually feels less painful. This allows you to take some much needed steps to helping that part of the body heal. Whether it be walking, stretching, exercises or physical therapy, with the assistance of cayenne, you can help your body heal a lot sooner!
Beyond the Cayenne
This rub is full of additional ingredients that will help your tired, achy muscles relax and maybe even heal some!!
Coconut oil is a wonderful oil that nourishes the skin. It is antimicrobial so is a perfect base for this rub. Coconut oil has been shown to have its own pain reducing and anti-inflammatory properties. 3
Raw cocoa butter is nourishing to the skin. It contains a high amount of antioxidants (just like chocolate) and will be nicely absorbed by the skin along with the beneficial herbs to help penetrate the muscles below.
Ginger is an spic with a long history of use in both Ayurvedic and Traditional Chinese Medicine. It is naturally anti-inflammatory and warming. When used in a muscle rub it will help stimulate the movement of blood flow. This allows for new blood to come into the area bringing with it new immune cells and helps push along old blood containing inflammatory cells that have done their job at the site of pain. 4
Turmeric, similar to ginger, is a spice with a very long history of use for its anti-inflammatory and healing effects. Turmeric will help to reduce swelling and keep the body’s inflammation in check. Remember inflammation is good for the healing process. But not when the body is chronically inflamed. 5
Peppermint Essential Oil has menthol in it. This is another common compound used for muscle soreness as it will reduce pain and relax the muscles. Peppermint cools an area providing similar relief as ice. 6 There is a nice balance here with the ginger and the peppermint. The former is working to warm the area and bring new blood flow and immune cells, while the later is constricting the muscles and moving the blood along to get rid of waste.
It is a really wonderful combination!
In addition to using the rub for sore muscles, remember to:
- Rest the area. One of the worst things most people do is to not sufficiently rest an area that chronically has pain. Rest is paramount to healing! With low back pain, lying down or sitting with legs up is often helpful.
- Drink plenty of water. Water is needed to carry your blood around and in that blood there are immune cells just waiting to help reduce inflammation. Adequate water is paramount to healing. Be sure to drink ½ your body weight in ounces of water a day. If you are dealing with an injured area, another 15-20 ounces will go a long way.
- Consider taking Turmeric capsules for arthritis pain reduction and healing. Turmeric capsules, 1500mg/day, has been shown to be just as effective as NSAIDs for reducing pain in folks with knee osteoarthritis, and without the side effects like NSAIDS (one of which is delayed healing). 5
Cayenne Muscle & Joint Rub Recipe:
- ¼ cup coconut oil
- ¼ cup raw cocoa butter (if you can’t find this, then just use all coconut oil)
- ¼ cup beeswax (grated to facilitate melting process)
- 2 tsp organic Cayenne powder
- 1 tsp organic Turmeric powder
- 1 tsp organic Ginger powder
- 10 drops of peppermint essential oil
- 4 oz. glass jar
- Place the coconut oil, cocoa butter and beeswax into a pyrex glass container and place that into a sauce pan that contains 2-3 inches of water (the pyrex will be resting in the water).
- Bring the water to a simmer. Continually stir the mixture until the cocoa butter and beeswax are completely melted, along with the coconut oil.
- Add the cayenne, turmeric and ginger powder and stir until they are completely moistened (roughly a minute).
- Remove from heat and add the peppermint essential oil.
- Pour the mixture into a glass jar immediately and allow to cool before placing a lid on top.
- Use liberally on affected area.
Storage: best stored in the fridge due to the fact it does not have preservatives in it. Lasts approximately 6-8 weeks.
CAUTION: be sure to wash your hands after you apply to affected area, as both cayenne and peppermint EO can be very strong. Also, turmeric may cause mild skin yellowing which is normal.
—-> Nutzt ihr Löwenzahnwurzeln? Wie und wofür?
Schon der Name verrät es: Auf Latein heißen Leinsamen “Linum usitatissimum” – übersetzt bedeutet dies “nützlicher Leinen”. Wer hätte gedacht, dass in dieser Pflanze so viel Energie drin steckt?
Aufgrund ihrer besonderen Beschaffenheit und Inhaltsstoffe können Leinsamen vielen Krankheiten vorbeugen und Beschwerden lindern. Von Verstopfung bis hin zu Herzproblemen.
Gleichzeitig sind Leinsamen eine super Unterstützung für alle, die Diät machen. Der Grund dafür sind die reichlich enthaltenen Ballaststoffe und guten Fette, die dazu führen, dass du schneller satt wirst und deine Verdauung und Stoffwechsel angeregt werden. Das hilft beim Abnehmen.
Die gesundheitsförderliche Wirkung lässt sich auf die drei Hauptinhaltsstoffe zurückführen: Omega-3-Fette (eine der höchsten Konzentrationen überhaupt), mehrfach ungesättigte Fettsäuren und Lignane. Diese Stoffe sind gut für Gelenke, Gedächtnis und die Abwehrkräfte. Dazu kommen noch zahlreiche Vitamine und Mineralien wie B-Vitamine, Kalzium, Zink, Eisen und noch vieles mehr. Um wirklich das meiste aus den Leinsamen herauszuholen, solltest du sie mahlen (geht sehr gut in einer Kaffeemühle oder einem Mixer). Mindestens 30 Minuten bevor du sie isst, solltest du sie dann in Wasser einweichen und anschließend gut abtropfen lassen.
Wenn du sie trocken und lichtgeschützt lagerst, halten sie sich bis zu ein paar Monaten. So hast du immer einen schnell verfügbaren Vorrat dieses großartigen Lebensmittels.
Am Besten schmecken sie in selbst gebackenem Brot oder als Zutat in deinem Müsli. So machen sie länger satt, schmecken gut und lassen sich natürlich super mit Früchten kombinieren.
Innerhalb von nur zwei Wochen wirst du Ergebnisse sehen können. Bessere Haut, bessere Verdauung und mehr Energie den ganzen Tag über. Jeder sollte diese nützlichen Samen in seinen Speiseplan einbauen. Damit tust du dir richtig gut.
Garlic was the miracle cure of ancient times, and now science has proved it to be incredibly beneficial. You won’t believe what this garlic and honey combination can heal! For immunity, heart health, digestion, and more, try out this recipe.
Garlic was the miracle cure of ancient times, and now science has proved it to be incredibly beneficial. You won’t believe what this garlic and honey combination can heal! For immunity, heart health, digestion, and more, try out this recipe.
Garlic is one of the ingredients that are most often used in cuisine around the world. It gives good taste to the food, but also is very powerful and can heal many ailments.
Consuming it raw can be an amazing and powerful medicine.
Garlic can decrease high blood pressure and cholesterol, prevent coronary heart disease and heart attack. Also, it can stop the effects of atherosclerosis.
If you are suffering from everyday health problems like hayfever, traveller’s diarrhea, cold, flu, bug bites and fungal infections, a garlic bulb can help you with it.
Moreover, garlic can help you manage the symptoms of osteoarthritis, diabetes, and an enlarged prostate.
Garlic can improve your immunity and the body’s ability to remove toxins. When it is combined with onion and ginger, it can aid you detox from chemotherapy.
How to use garlic?
The best way to consume garlic is raw (not from China), because allicin, its active ingredient is decentralized by heat. Crush and slice the clove and leave it for 15 minutes before consuming it. When you crush the bulp, it activates a reaction that makes alicin more bio available.
It should be eaten on an empty stomach because if the stomach is overwhelmed with food, it has difficulties to process and absorb all the food’s nutrients.
RAW GARLIC AND HONEY
Chop 2-3 garlic cloves into small pieces. Mix them with a tablespoon of raw honey.
If you take this combination every day, you will feel more energized and healthy.
GARLIC FLU TONIC
Wear gloves if you have sensitive skin and don’t rub your eyes when you make this tonic because some of the natural oils can trigger a skin rash or burn.
- Roughly chopped half yellow onion
- 5 roughly chopped garlic cloves
- 2 red chili peppers, also roughly chopped
- 1 tablespoon of chopped ginger
- Squeezed juice of 1 lemon
- Raw and unfiltered apple cider vinegar (buy here)
How to prepare it?
In a mason jar of 350 ml to 500 ml place the roughly chopped onion. Next, add the chopped garlic. Follow with the red chili peppers, seeds and all.
Then, carefully place some chopped ginger. Squeeze the lemon in a separate bowl, throw away the seeds and then pour the juice.
At the end add the apple cider vinegar and leave about a cm of room at the top. Close the jar and place it on the counter or in a pantry.
Now you can start using the tonic remedy for sore throat, cold, and flu.
Originally taken from My Life Style
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